Note: Beginning with Fall 2016, The Basic Science program has been combined into the Mentored Scientist Award mechanism - which now has three scientific priority areas for funding: Basic Science, Clinical/ Translational/ Epidemiological/ Behavioral, and Implementation Science.
Below are the awards made under the Basic Science program from 1994 to 2016.
58 Awards
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The Role of Autophagy in HIV Infection
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Identification of Novel HIV Co-Receptors and Characterization of Their Role in Viral Transmission
Identification of Novel HIV Co-Receptors and Characterization of Their Role in Viral Transmission
Abstract
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The Role of Autophagy in HIV Infection
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Characterization of the Epstein Barr Virus BMRF-2 Gene and its Role in the Pathogenesis of Hairy Leukoplakia
Characterization of the Epstein Barr Virus BMRF-2 Gene and its Role in the Pathogenesis of Hairy Leukoplakia
Abstract
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Characterization of the Epstein Barr Virus BMRF-2 Gene and its Role in the Pathogenesis of Hairy Leukoplakia
Characterization of the Epstein Barr Virus BMRF-2 Gene and its Role in the Pathogenesis of Hairy Leukoplakia
Abstract
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Investigation of the Effects of IL-2 on the Frequency of FcRL3+ Tregs and T cell Activation in HIV-infected Patients
Investigation of the Effects of IL-2 on the Frequency of FcRL3+ Tregs and T cell Activation in HIV-infected Patients
Abstract
Regulatory T cells (Tregs), defined by expression of FoxP3 and the IL-2 receptor CD25, play a critical role in self-tolerance and suppressing immune responses elicited against infectious agents such as HIV. As it is now recognized that some of the immunopathology wrought by HIV infection may be due to aberrant immune activation, much focus is centered on the role of Tregs and how they may decrease immune activation and slow disease progression.
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Biochemical and Clinical Consequences of Antiviral Resistance Mutations in HIV-1 Protease
Biochemical and Clinical Consequences of Antiviral Resistance Mutations in HIV-1 Protease
Abstract
Publications:
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BRD4 Overexpression Increases the Propensity for Proviral Latency and Determine how this Relates to HIV Latency in vivo.
BRD4 Overexpression Increases the Propensity for Proviral Latency and Determine how this Relates to HIV Latency in vivo.
Abstract
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Pilot Study of HIV-1-Specific Immunity Following Antiretroviral Treatment Interruption
Pilot Study of HIV-1-Specific Immunity Following Antiretroviral Treatment Interruption
Abstract
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Pilot Study of HIV-1-Specific Immunity Following Antiretroviral Treatment Interruption
Pilot Study of HIV-1-Specific Immunity Following Antiretroviral Treatment Interruption
Abstract